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Depolarization as a regulatory signal at voltage-gated calcium channels.

   
@article{APS6399,
	author = {David J. Triggle},
	title = {Depolarization as a regulatory signal at voltage-gated calcium channels.},
	journal = {Acta Pharmacologica Sinica},
	volume = {17},
	number = {3},
	year = {2016},
	keywords = {},
	abstract = {Changes in membrane potential are a regulatory signal for voltage-gated ion channels including the family of Ca2+ channels. This regulatory role includes the voltage-dependent opening, closing, and inactivation of ion channels and the control of drug access and affinity for discrete channels states. Membrane potential is both a short- and long-term regulatory signal controlling the number and function of these voltage-gated channels. Depolarization of neuronal and neurosecretory cells produces down-regulation of L-type voltage-gated Ca2+ channels. These processes are described and their relevance to physiologic and pathologic processes of neuronal development and neuroprotection are indicated.},
	issn = {1745-7254},	url = {http://www.chinaphar.com/article/view/6399}
}